Search results for " Delayed [Medical Subject Headings]"

showing 10 items of 151 documents

Serviceability of segmental concrete arch-frame bridges built by cantilevering

2013

The evaluation of the safety degree in service life for a segmental prestressed concrete bridge built by the cantilever method presents different aspects related to strength, deformability, durability and to effects of time-dependent phenomena as creep and shrinkage. The case-study of a prestressed concrete frame bridge with inclined piers is presented, built by cantilevering, with precast segments. By considering the fundamental items of design and construction stages, the implications due to the arch effect in this static scheme are investigated, with respect to an equivalent bridge with a final scheme of continuous girder on vertical piers. The birth of a significant value of axial force…

EngineeringServiceability (structure)business.industryBuilding and ConstructionStructural engineeringSegmental bridge arch-frame bridge cantilever construction serviceability durability limit state delayed deformationsDurabilitylaw.inventionSettore ICAR/09 - Tecnica Delle CostruzioniPrestressed concretelawPrecast concreteGirderCantilever methodLimit state designGeotechnical engineeringArchbusiness
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Maternal seafood consumption during pregnancy and child attention outcomes: a cohort study with gene effect modification by PUFA-related genes

2019

Abstract Background There is a need to test the fetal programming theoretical framework in nutritional epidemiology. We evaluated whether maternal seafood intake during pregnancy was associated with 8-year-old attention outcomes after adjusting for previous child seafood intake and cognitive function. We also explored effect modification by several single nucleotide polymorphisms (SNPs) related with polyunsaturated fatty acid (PUFA) metabolism. Methods Our final analyses included 1644 mother-child pairs from the prospective INMA (INfancia y Medio Ambiente) cohort study (Spain, recruitment between 2003 and 2008). We used food frequency questionnaires to assess prenatal and postnatal seafood …

EpidemiologySingle-nucleotide polymorphism03 medical and health sciences0302 clinical medicinePregnancyEnvironmental healthmedicineHumansAttention deficit hyperactivity disorderProspective Studies030212 general & internal medicineChildchemistry.chemical_classificationPregnancyNutritional epidemiologybusiness.industryIncidence (epidemiology)food and beveragesGeneral Medicinemedicine.diseaseDietSeafoodchemistryAttention Deficit Disorder with HyperactivitySpainPrenatal Exposure Delayed EffectsRelative riskFatty Acids UnsaturatedFemalebusiness030217 neurology & neurosurgeryCohort studyPolyunsaturated fatty acidInternational Journal of Epidemiology
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Prenatal diazepam exposure functionally alters the GABA(A) receptor that modulates [3H]noradrenaline release from rat hippocampal synaptosomes.

2002

In rats, exposure to diazepam (DZ) during the last week of gestation is associated with behavioral alterations (in some cases sexually dimorphic) that appear when the animals reach adulthood. This study was conducted to evaluate the effects of prenatal DZ exposure on the function of the gamma-aminobutyric (GABA)(A) receptor complex. The method used - perfusion of rat hippocampal nerve terminals labeled with [3H]noradrenaline (NA) - allowed us to evaluate the effects of DZ on a specific native GABA(A) receptor subtype which is located on hippocampal noradrenergic nerve endings and mediates the release of NA. Muscimol stimulated synaptosomal release of [3H]NA in a concentration-dependent mann…

Fetal ProteinsMaleBaclofenNerve Tissue ProteinsPregnanoloneBicucullinein uteroHippocampusGABA AntagonistsNorepinephrineAllosteric RegulationPregnancyAnimalsPicrotoxinRats WistarGABA AgonistsDiazepam In utero [3H]Noradrenaline release Synaptosomes GABAA receptor Allosteric modulationallosteric modulationDiazepamMental DisordersGABAA receptorReceptors GABA-ARatsProtein SubunitsPrenatal Exposure Delayed EffectsSettore BIO/14 - FarmacologiaFemaleSynaptosomesDevelopmental neuroscience
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Prenatal low-level exposure to CO alters postnatal development of hippocampal nitric oxide synthase and haem-oxygenase activities in rats.

2001

The effects of prenatal CO exposure (150 ppm from days 0 to 20 of pregnancy) on the postnatal development of hippocampal neuronal NO synthase (nNOS) and haem-oxygenase (HO-2) isoform activities in 15-, 30- and 90-d-old rats were investigated. Unlike HO-2, hippocampal nNOS activity increased from postnatal days 15-90 in controls. Prenatal CO produced a long-lasting decrease in either nNOS or HO-2. The results suggest that the altered developmental profile of hippocampal nNOS and HO-2 activities could be involved in cognitive deficits and long-term potentiation dysfunction exhibited by rats prenatally exposed to CO levels resulting in carboxyhaemoglobin (HbCO) levels equivalent to those obser…

Gene isoformmedicine.medical_specialtyNitric Oxide Synthase Type IHippocampal formationHippocampusCarbon monoxide; haem-oxygenase; hippocampus; nitric oxide synthase; prenatal exposure.HemoglobinsPregnancyInternal medicinemedicineAnimalsPharmacology (medical)Rats WistarPharmacologyDevelopmental profilePregnancyCarbon MonoxidebiologyChemistryLong-term potentiationLow level exposuremedicine.diseaseHaem OxygenaseRatsNitric oxide synthaseIsoenzymesPsychiatry and Mental healthEndocrinologyPrenatal Exposure Delayed EffectsHeme Oxygenase (Decyclizing)biology.proteinFemaleNitric Oxide SynthaseThe international journal of neuropsychopharmacology
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Association of maternal prenatal smoking GFI1-locus and cardio-metabolic phenotypes in 18,212 adults

2018

Background: DNA methylation at the GFI1-locus has been repeatedly associated with exposure to smoking from the foetal period onwards. We explored whether DNA methylation may be a mechanism that links exposure to maternal prenatal smoking with offspring's adult cardio-metabolic health.Methods: We meta-analysed the association between DNA methylation at GFI1-locus with maternal prenatal smoking, adult own smoking, and cardio-metabolic phenotypes in 22 population-based studies from Europe, Australia, and USA (n= 18,212). DNA methylation at the GFI1-locus was measured in whole-blood. Multivariable regression models were fitted to examine its association with exposure to prenatal and own adult s…

Genetics and Molecular Biology (all)MaleNetherlands Twin Register (NTR)0301 basic medicineResearch paperGFI1 protein humanGFI1-locusraskausResearch & Experimental Medicinecardio-metabolic phenotypesBiochemistryEpigenesis GeneticGLOBAL Meth QTL Consortium0302 clinical medicinePregnancySmoke030212 general & internal medicinematernal prenatal smokingDNA METHYLATIONmedia_commonRISK2. Zero hungereducation.field_of_studySmokingta3142General MedicineMiddle Agedgenetics [Transcription Factors]3. Good healthDNA-Binding ProteinsPhenotypeMedicine Research & ExperimentalCARDIOVASCULAR-DISEASEepigenetiikkaPopulation SurveillancePrenatal Exposure Delayed EffectsDNA methylation/dk/atira/pure/sustainabledevelopmentgoals/good_health_and_well_beingFemaleDisease SusceptibilityBIOS ConsortiumMedical GeneticsLife Sciences & BiomedicineAdultmedicine.medical_specialtyOffspringBirth weightPopulationMothersgenetics [DNA-Binding Proteins]ta3111MethylationGeneral Biochemistry Genetics and Molecular BiologyDIET03 medical and health sciencesMedicine General & InternalSDG 3 - Good Health and Well-beingtupakointiGeneral & Internal MedicineInternal medicine/dk/atira/pure/keywords/cohort_studies/netherlands_twin_register_ntr_medicinemedia_common.cataloged_instanceHumansddc:610adverse effects [Maternal Exposure]EXPOSUREEpigeneticsEuropean unioneducationMedicinsk genetikEPIGENOME-WIDE ASSOCIATIONPregnancyBiochemistry Genetics and Molecular Biology (all)Science & Technologybusiness.industryadverse effects [Smoking]DNA Methylationta3121medicine.diseaseBIRTH-WEIGHT030104 developmental biologyEndocrinologyGenetic Locisydän- ja verisuonitauditCpG IslandsCIGARETTE-SMOKINGCESSATIONEnergy Metabolismmetabolism [Myocardium]businessBody mass indexBiomarkersTranscription FactorsEBioMedicine
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Gestational exposure to cocaine alters cocaine reward

2006

Exposure of the developing foetus to drugs of abuse during pregnancy may lead to persistent abnormalities of brain systems involved in drug addiction. Mice prenatally exposed to cocaine (25 mg/kg), physiological saline or non-treated during the last 7 days of pregnancy were evaluated in adulthood for the rewarding properties of cocaine (3, 25 and 50 mg/kg), using the conditioned place preference procedure. Dams treated with physiological saline gained significantly less weight over the course of gestation than controls; no other differences were observed in the maternal and offspring data. All the animals developed preference to 3 and 25 mg/kg of cocaine, but those treated prenatally with c…

Gestational exposureOffspringmedia_common.quotation_subjectPhysiologySocial EnvironmentChoice BehaviorCocaine-Related DisordersMiceCocaineRewardPregnancyOrientationConditioning PsychologicalAvoidance LearningmedicineAnimalsmedia_commonPharmacologyMotivationFetusPregnancyDose-Response Relationship DrugAddictionAssociation LearningBrainmedicine.diseaseConditioned place preferencePsychiatry and Mental healthPrenatal Exposure Delayed EffectsAnesthesiaGestationFemaleBrain stimulation rewardCuesPsychologyBehavioural Pharmacology
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Non-alcoholic fatty pancreas disease pathogenesis: a role for developmental programming and altered circadian rhythms.

2013

Objectives Emerging evidence suggests that maternal obesity (MO) predisposes offspring to obesity and the recently described non-alcoholic fatty pancreas disease (NAFPD) but involved mechanisms remain unclear. Using a pathophysiologically relevant murine model, we here investigated a role for the biological clock - molecular core circadian genes (CCG) in the generation of NAFPD. Design Female C57BL6 mice were fed an obesogenic diet (OD) or standard chow (SC) for 6 weeks, prior to pregnancy and throughout gestation and lactation: resulting offspring were subsequently weaned onto either OD (Ob_Ob and Con_Ob) or standard chow (Ob_Con and Con_Con) for 6 months. Biochemical, pro-inflammatory and…

HeredityPhysiologylcsh:MedicineCLOCK ProteinsGene ExpressionMouse ModelsGastroenterology and HepatologyResearch and Analysis MethodsModel OrganismsPregnancyGeneticsMedicine and Health SciencesAnimalsRNA MessengerObesitylcsh:ScienceNutritionAnalysis of Variancelcsh:RBody WeightGene Expression Regulation DevelopmentalPancreatic DiseasesBiology and Life SciencesAnimal ModelsCircadian RhythmMice Inbred C57BLPhysiological ParametersPrenatal Exposure Delayed Effectslcsh:QFemaleEpigeneticsAnatomyPhysiological ProcessesDigestive SystemChronobiologyResearch ArticlePloS one
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Reproductive medicine and inheritance of infertility by offspring: the role of fetal programming.

2011

Objective To summarize the molecular processes involved in fetal programming, to describe how assisted reproduction technologies (ART) may affect the epigenetic pattern of the embryo, and to highlight the current knowledge of the role of perinatal events in the subsequent development of reproductive pathology affecting infertile patients. Design A literature review of fetal programming of adulthood gynecologic diseases and ART. A Medline search was performed with the following keywords: (fetal programming OR epigenetics OR methylation OR acetylation) AND (IVF OR ART) AND (gynecology). Articles up to October 2010 were selected. Articles and recent reviews were classified by human and animals…

Infertilitymedicine.medical_specialtyOffspringPopulationMEDLINEReproductive medicineBioinformaticsEpigenesis GeneticPregnancymedicineHumansEpigeneticseducationGynecologyeducation.field_of_studyFetusbusiness.industryObstetrics and GynecologyDNA Methylationmedicine.diseaseReproductive MedicinePrenatal Exposure Delayed EffectsObservational studyFemalebusinessGenital Diseases FemaleInfertility FemaleFertility and sterility
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INFLUENCE OF LENGTH ON THE NOISE DELAYED SWITCHING OF LONG JOSEPHSON JUNCTIONS

2008

The transient dynamics of long overlap Josephson junctions in the frame of the sine-Gordon model with a white noise source is investigated. The effect of noise delayed decay is observed for the case of overdamped sine-Gordon equation. It is shown that this noise induced effect, in the range of small noise intensities, vanishes for junctions lengths greater than several Josephson penetration length.

Josephson effectPhysicsCondensed matter physicsJosephson phaseNoise inducedCondensed Matter - SuperconductivityApplied MathematicsFOS: Physical sciencesThermal fluctuationsJosephson energyWhite noisenoise delayed switchingSuperconductivity (cond-mat.supr-con)Pi Josephson junctionCondensed Matter::SuperconductivityModeling and SimulationEngineering (miscellaneous)International Journal of Bifurcation and Chaos
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Obesogen effects after perinatal exposure of 4,4′-sulfonyldiphenol (Bisphenol S) in C57BL/6 mice

2016

International audience; Bisphenol A were removed from consumer products and replaced by chemical substitutes such as Bisphenol S (BPS). Based on their structural similarity, BPS may be obesogen like Bisphenol A in mice. Our objective was to determine the impact of BPS on lipid homeostasis in C57B1/6 mice after perinatal and chronic exposure. Pregnant mice were exposed to BPS via the drinking water (0.2; 1.5; 50 mu g/kg bw/d). Treatment began at gestational day 0 and continued in offspring up to 23-weeks old. Then, offspring mice were fed with a standard or high fat diet. The body weight, food consumption, fat mass and energy expenditure were measured. A lipid load test was performed to chec…

Male0301 basic medicineLeptinBisphenol S[ SDV.TOX ] Life Sciences [q-bio]/ToxicologyAdipose tissue010501 environmental sciencesToxicologyurologic and male genital diseases01 natural sciencesPolyethylene GlycolsMicechemistry.chemical_compoundPregnancyInduced ObesityHyperinsulinemiapériode perinataleObesogenSulfones2. Zero hungerLeptinHigh-Fat Dietsanté humaineLipidsEnergy-Balance3. Good healthSafe AlternativesobésitéAdipose TissuePrenatal Exposure Delayed Effects[SDV.TOX]Life Sciences [q-bio]/Toxicologybisphénol sFemalehormones hormone substitutes and hormone antagonistsmedicine.medical_specialtyOffspringDiet High-Fat03 medical and health sciencesInsulin resistancePhenolsInternal medicinemedicineAnimalshoméostasie lipidiqueObesityRNA MessengerTriglycerides0105 earth and related environmental sciencesDose-Response Relationship DrugAdiponectinTriglycerideInsulin-ResistanceBody WeightOverweightmedicine.diseasebisphenol S;food contaminant;perinatal exposure;low dose;obesogenPerinatal exposureMice Inbred C57BLFood contaminant030104 developmental biologyEndocrinologycontaminant chimiqueLow doseGlucoseMetabolismGene Expression RegulationchemistryIn-VitroObesogenAnalogs
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